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Maternal hyperhomocysteinemia induces fetal growth restriction by suppressing angiogenesis at the maternal-fetal interface.

Cell Biosci · 2026 · PMC12882372 · PMID 41514375

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showed a trendno p-value reported
The KEGG pathway enrichment analysis of DEGs showed a trend of enrichment in lipid metabolism pathways, including cholesterol metabolism, PPAR signaling pathway, sphingolipid metabolism, ether lipid metabolism, and biosynthesis of unsaturated fatty acids (Fig. 4 C).

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