Macrophage surface protein Mac-2 mediates inflammatory and stromal stress pathways in doxorubicin-induced cardiac injury.

Their HW/BW ratio was elevated compared with WT controls and showed a trend toward significance versus the DOX WT [KO BM] group (Fig. 6 c, p = 0.05, n = 9–13).

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6 i–o, p = 0.05 , n = 8–13), along with % fibrosis increased in DOX WT [KO BM] compared to other dox groups, differences did not reach statistical significance (Fig. 6 p, n = 9–13).

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