Mycobiome dysbiosis and genetic predisposition to elevated IL-17A contribute to fibrosis in MASLD.

Although intrahepatic IL-17A data for patients with MASLD included in the gut mycobiome analysis were available for only a small subset of the cohort, the results showed a trend toward a positive correlation between gut mycobiome dysbiosis and increased hepatic IL-17A levels (Spearman correlation = 0.352, p = 0.218).

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Finally, an increasing trend in CTG species abundance was also observed when samples were stratified by histology-defined fibrosis stage ( Fig.

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