Functional remodeling of iNKT cells by sulfatide-reactive type II NKT cells reprograms alveolar macrophages to alleviate lung ischemia-reperfusion injury.

Although the proportion and number of IL-4- and IL-17A-producing iNKT cells showed a decreasing trend, this change was not statistically significant (Fig. 5E–H, K ).

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In normoxia, IL-10 similarly enhanced Arg-1 mRNA expression, with a trend toward increased CD206 mRNA, MFI for CD206, and Arg-1 immunofluorescence, although these changes did not reach statistical significance (Figs. 7C and 8P–R ).

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