Scaffold-mediated miRNA-155 inhibition promotes regenerative macrophage polarisation leading to anti-inflammatory, angiogenic and neurogenic responses for wound healing.

2 J), further supported macrophage polarisation toward an anti-inflammatory phenotype on CG-155-i scaffolds, as evidenced by an increasing trend in CD206 expression and a ∼3-fold upregulation of ARG1.

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3 F) in the CG-155-i group relative to CG scaffolds, although this did not reach statistical significance.

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In contrast, CD86 expression showed a decreasing trend in both nanoparticle-functionalised groups compared with gene-free scaffolds.

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