Organ-specific immune responses are strain-dependent in a mouse model of <i>Cryptococcus neoformans</i> brain infection.

In contrast, there was a significant correlation between fungal brain burdens and numbers of macrophages, and a positive trend between neutrophils and Ly6C hi monocytes that did not quite reach statistical significance, indicating that inflammatory responses measured by proliferation and/or recruitment of these cells are likely influenced by fungal brain burden ( Fig. 3B ).

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neoformans strain used although there was a slight trend of reduced CD44 expression during infection with Zc1 and Zc15 ( Fig. 6B ), which largely mimicked the pattern seen in brain fungal burdens ( Fig. 1C ).

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In contrast, there was a significant correlation between fungal brain burdens and numbers of macrophages, and a positive trend between neutrophils and Ly6C hi monocytes that did not quite reach statistical significance, indicating that inflammatory responses measured by proliferation and/or recruitment of these cells are likely influenced by fungal brain burden ( Fig. 3B ).

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There was less recruitment of CnT.II cells in the Zc15-infected brains compared with H99, although this did not reach statistical significance ( Fig. 7B ).

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