Reversing T cell dysfunction in a novel <i>in vitro</i> model of T cell exhaustion reveals differential roles of RASA2.

Analysis of the secretory profile showed that the significantly increased Granzyme A secretion of Tex cells compared to Ts was maintained after resting, while numerical reductions in IL-2 secretion (0.45-fold) of Tex cells was also observed albeit this did not reach statistical significance ( Supplementary Figure S7 ).

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