Blockade of Presynaptic α<sub>2</sub>δ<sub>1-2</sub> Subunits of Voltage-Gated Ca²⁺ Channels Attenuates Neurobiochemical and Sensorimotor Deficits After Traumatic Brain Injury in Mice.

Neurochem Res · 2026 · PMC13018067 · PMID 41880029

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did not reach statistical significanceno p-value reported

Moreover, protein levels of the α 2 δ 2 subunits of VGCCs in both pre- and postsynaptic terminals were higher in the TS group compared with CS and C60 groups by 27.23% and 24% respectively, although this difference did not reach statistical significance (Fig. 4 C; CS vs.

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Blockade of Presynaptic α&lt;sub&gt;2&lt;/sub&gt;δ&lt;sub&gt;1-2&lt;/sub&gt; Subunits of Voltage-Gated Ca²⁺ Channels Attenuates Neurobiochemical and Sensorimotor Deficits After Traumatic Brain Injury in Mice.

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Blockade of Presynaptic α<sub>2</sub>δ<sub>1-2</sub> Subunits of Voltage-Gated Ca²⁺ Channels Attenuates Neurobiochemical and Sensorimotor Deficits After Traumatic Brain Injury in Mice.