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Ursodeoxycholic Acid Attenuates Lipopolysaccharide-Induced Myocardial Injury by Inhibiting Oxidative Stress, Inflammation, and Apoptosis: The Interplay of Sirt1/Nrf2 and Akt/NF-κB Signaling Pathways.

Int J Mol Sci · 2026 · PMC13026358 · PMID 41898702

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CAT activity was decreased in the LPS-treated group ( p < 0.05) compared with the control group, while pretreatment with UDCA partially restored CAT activity, although the difference between the LPS and UDCA + LPS groups did not reach statistical significance.

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