The Therapeutic Administration of <i>Lactobacillus brevis</i> ZG2488 Suppresses Influenza A Virus Replication Through a Viability-Dependent Host Transcriptional Modulation Mechanism.

At 24 h post-infection, the qPCR quantification of viral load revealed a highly significant reduction in viral load compared with the virus control group (PR8-V) for both pretreatment durations ( p < 0.001).

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brevis ZG2488-treated mice showed a downward trend compared with the infection control group, although the differences did not reach statistical significance ( Figure 4 D,E).

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