Circulating sCD163 concentration was negatively correlated with LVEF ( p = .01) and showed a trend towards a negative correlation with NT‐proBNP, indicating that higher sCD163 levels are associated with more severe systolic dysfunction (Figure 1C,D ).
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Macrophage Cluster of Differentiation 163 promotes post-infarction cardiac repair and preserves left ventricular function via osteopontin.
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