Theaflavin-3,3'-Digallate Targets Pin1 to Suppress Hepatocellular Carcinoma Malignant Proliferation Through Modulation of MAPK and PI3K/AKT Signaling Pathways In Vitro.

Figure 6 H and Figure S6 indicates that the total protein expression of JNK remains stable, while the expression level of its phosphorylated form p-JNK showed a trend of first increasing and then decreasing, suggesting that the JNK MAPK pathway may be instantaneously activated during TF3 treatment.

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The p values between 0.01 and 0.05 were regarded as significant difference, while p values less than 0.01 were regarded as highly significant differences.

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