Hyaluronidase-1 mediates postprandial suppression of hepatic gluconeogenesis.

Life Metab · 2025 · PMC13122362 · PMID 42058136

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First, hepatocytes isolated from overnight-fasted mice exhibited increased glucose output only in response to glucagon and dibutyryl-cAMP (db-cAMP), a membrane-permeable analog of cAMP, while HYAL1 showed a trend towards suppressing gluconeogenesis in both conditions.

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