Purinergic signaling modulates endothelial-to-mesenchymal transition in human aortic valve endothelial cells.

VE-cadherin expression was decreased as well by CGS treatment (p < 0.05) but failed to reach statistical significance with BAY treatment ( Fig. 3 C). 3.4 TGFβ stimulation, inhibition of CD39 or treatment with BzATP enhances p38 phosphorylation in VEC Activation of p38 MAPK (p38) has been linked to TGFβ1-induced EMT in human renal epithelial cells [ 34 ] as well as to EndMT in human dermal microvascular endothelial cells [ 35 ].

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