L-carvone protects against myoglobinuric acute tubular necrosis via inhibition of NF-κB and caspase-dependent apoptotic pathways.

While the low-dose L -carvone (25 mg/kg) treatment reduced IL-1β levels to 4.21, this decrease did not reach statistical significance against the induction group (p = 0.3024).

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Specifically, the induction model successfully elicited massive, characteristic spikes in blood urea nitrogen (BUN) exceeding 100 mg/dL (mean 108.00 mg/dL), along with highly significant elevations in serum creatinine.

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