Nuclear protein 1 is a cell death regulator in primary human airway epithelial cells and reduced in idiopathic pulmonary fibrosis.

5 K) and, even though early and late apoptosis were not significantly upregulated individually, as monitored by Annexin V/PI, both showed an increasing trend after NUPR1 inhibition (Fig. 5 L, Supplemental Fig.

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The quantification of club and basal cells showed a trend towards an increase in IPF-derived cells, which, however, failed to reach significance (Supplemental Fig.

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Western blot analysis revealed very heterogenous protein levels of NUPR1 with a trend towards lower abundance in IPF-derived phBECs which, however, failed to reach significance.

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