A novel bile salt hydrolase-producing <i>Ligilactobacillus salivarius</i> prevents diet-induced obesity via regulation of bile acid metabolism and glucagon-like peptide 1 restoration.

Gut Microbes · 2026 · PMC13166280 · PMID 42092775

Europe PMC DOI

hedged sentence

0.0860

closest p · 1.7× alpha

0.0860

boldest claim

The sentences

did not reach statistical significancep = 0.086so close (0.05 < p ≤ 0.1)

Moreover, TUDCA and GUDCA also showed trends toward enrichment in the liver, although the differences did not reach statistical significance ( Figure 7G-H , p = 0.086 and p = 0.073, respectively).

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Quoted from the open-access full text in Europe PMC under the licence the publisher applied. The sentence is reproduced exactly as published; the emphasis is ours.

A novel bile salt hydrolase-producing &lt;i&gt;Ligilactobacillus salivarius&lt;/i&gt; prevents diet-induced obesity via regulation of bile acid metabolism and glucagon-like peptide 1 restoration.

The sentences

A novel bile salt hydrolase-producing <i>Ligilactobacillus salivarius</i> prevents diet-induced obesity via regulation of bile acid metabolism and glucagon-like peptide 1 restoration.