NDUFA9 expression showed a trend of increasing with advancing pathological T stage, with a statistically significant increase observed between T1 and T2 stages (Fig. 1C ).
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YY1-mediated NDUFA9 upregulation promotes NSCLC cell growth through mitochondrial and Akt-mTOR pathway modulation.
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The stringent intersection converged on a single, highly significant co-expressed molecule: NDUFB9 (Fig. 5L ).