Sustained NPSLE-like phenotype in the absence of systemic lupus-like disease in TLR7-deficient B6.Nba2 mice.

Further analyses of TLR4, TLR7 and TLR9 protein expression by splenic pDCs, cDCs and B cells showed that TLR9 was specifically upregulated on cDCs, while TLR4 was found to be elevated on pDC, cDC, and B cells, although the data did not reach statistical significance ( Fig. 6D – G ).

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