Engineered Nestin<sup>+</sup> TSPC-Derived Exosomes Promote Tendon Repair Via Metabolic Reprogramming.

Nes‐EVs elicited a more pronounced reduction in γ‐H2AX compared to Con‐EVs and similarly decreased p16 INK4A levels, although the latter did not reach statistical significance (Figure 4j–m ).

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