Nonlinear combinatorial analysis of blood transcriptomes identifies PRKAR1A as a regulator of TDP-43 pathophysiology in amyotrophic lateral sclerosis.

Biol Methods Protoc · 2026 · PMC13293084 · PMID 42359392

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The expression levels of PRKAR1A and QPCT were significantly higher in ALS than in healthy controls, whereas TMEM71 showed a trend toward higher expression in ALS ( Fig. 6b ).

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