Barely Significant
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Müller Glia-Exclusive CLRN1 Expression Drives Non-Cell-Autonomous Photoreceptor Degeneration in Usher Syndrome Type 3A.

Invest Ophthalmol Vis Sci · 2026 · PMC13332521 · PMID 42390169

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nominally significantP < 0.05actually significant
GO analysis of nominally significant genes ( P < 0.05) revealed coherent downregulation of glutamate receptor signaling (adjusted P = 0.031), receptor localization to synapses (adjusted P = 0.031), and GABAergic transmission regulation (adjusted P = 0.034), indicating progressive failure of Müller glia synaptic support functions ( Fig. 6 a; Supplementary Datasets S2 , S3 ).

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