Integrated computational analysis identifies FABP4, PTGS2, and HPGD as Key molecular targets linking PET microplastic exposure to metabolic dysfunction-associated steatotic liver disease.

Apart from these relationships, the correlations between most T cell subpopulations and their association with myeloid cells were close to zero and weakly significant, suggesting that the primary covariation in immune infiltration is concentrated in the myeloid/intrinsic immune components and the opposing changes between the “resting-activated” states.

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