Latency associated peptide has in vitro and in vivo immune effects independent of TGF-beta1.

CD36 appeared to play less of a role in this immunologic effect as anti-CD36 antibodies only showed a trend toward interference with LAP-mediated inhibition of DTHR, and the reversal appeared incomplete [DTH thickness 61± 2×10 −4 in (control) vs. 19±3×10 −4 in (LAP alone) vs. 28±2×10 −4 in (LAP+ anti-CD36 Ab), p = 0.058].

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