Hypothermia and postconditioning after cardiopulmonary resuscitation reduce cardiac dysfunction by modulating inflammation, apoptosis and remodeling.

These results, however, did not reach statistical significance possibly due to an inadequate sample size rather than a true difference was absent.

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Very recently, a retrospective subgroup analysis of the ‘Hypothermia after Cardiac Arrest’ trial revealed a favorable trend for early cooling in patients with cardiac arrest following myocardial ischemia [23] .

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