lwoffii F78, respectively), but these differences did not reach statistical significance.
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Maternal TLR signaling is required for prenatal asthma protection by the nonpathogenic microbe Acinetobacter lwoffii F78.
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lwoffii F78–treated mother mice showed a significant down-regulation of TLR6 and 7 mRNA (compared with sham-treated controls), accompanied by a nonsignificant trend toward lower mRNA levels in all other TLRs tested ( Fig. 4 B ).