MSH2/MSH6 complex promotes error-free repair of AID-induced dU:G mispairs as well as error-prone hypermutation of A:T sites.

There was a less robust, though still significant, decrease in mutations at T residues and T W motifs in the Jh2-Jh4 region, but this was only marginally significant in the V186.2 region, most likely due to the low number of mutations at T sites.

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