This relationship is in line with lesions AMY failing to increase feeding bouts, which strengthens the failure of hamsters to display any eating stimuli aside prior to awakening state (6 th -7 th day) in which CPP-treated hibernators displayed moderately significant increases of feeding intervals [ 36 ].
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Amygdalar glutamatergic neuronal systems play a key role on the hibernating state of hamsters.
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