Familial amyloid precursor protein mutants cause caspase-6-dependent but amyloid β-peptide-independent neuronal degeneration in primary human neuron cultures.

A β 1−42 increased levels of beading in pBudEGFP/APP MV -transfected neurons after 24 h of incubation, but the increase did not reach statistical significance compared with A β 42−1 ( Figure 4c ).

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