Endoplasmic reticulum stress inhibits STAT3-dependent suppression of hepatic gluconeogenesis via dephosphorylation and deacetylation.

Nonfasting blood glucose levels 14 days after treatment with PBA tended to be lower in PBA-treated db/db mice (381 ± 57 mg/dL; n = 6) than in untreated db/db mice (474 ± 84 mg/dL; n = 6), although the tendency did not reach statistical significance.

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