Spatio-temporal course of macrophage-like cell accumulation after experimental embolic stroke depending on treatment with tissue plasminogen activator and its combination with hyperbaric oxygenation.

Taking these results together with our previous work that showed a trend to worsened long-term clinical outcome (as defined by a combined endpoint of neurological deficit and premature death) in tPA+HBO-treated subjects vs the control and tPA group, 50 the inhibition of macrophage-like accumulation in the ischaemic area has to be discussed in a critical manner.

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