Negative regulation of pulmonary Th17 responses by C3a anaphylatoxin during allergic inflammation in mice.

Although the C3aR −/− mice treated with anti-IL-17 showed slightly lower AHR compared with isotype-treated mice, it did not reach statistical significance ( Figure 7B ), suggesting little role of IL-17 in regulating airway reactivity in C3aR −/− mice.

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