Lack of transient receptor potential vanilloid 1 channel modulates the development of neurogenic bladder dysfunction induced by cross-sensitization in afferent pathways.

The normalized amplitude of the contractile response to KCl was 22.7 ± 3.3 g/g in TRPV1 −/− group, which was lower than in WT group (44.5 ± 12.2 g/g), however, this difference did not reach statistical significance ( P = 0.12 to WT, Figure 3D ).

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