There was a slightly excess of heterozygotes and a deficit of GG homozygotes among CAD patients in the North-American cohort, resulting in a nominally significant deviation from Hardy-Weinberg equilibrium (HWE) in this group (Table 3 footnote).
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Association of circulating levels of nicotinamide phosphoribosyltransferase (NAMPT/Visfatin) and of a frequent polymorphism in the promoter of the NAMPT gene with coronary artery disease in diabetic and non-diabetic subjects.
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