Indeed, a clear trend toward less-activated immune cells had surfaced in hAAT-treated CP-operated animals.
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Human α1-Antitrypsin Binds to Heat-Shock Protein gp96 and Protects from Endogenous gp96-Mediated Injury In vivo.
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Indeed, a clear trend toward less-activated immune cells had surfaced in hAAT-treated CP-operated animals.