Toll-like receptor signaling adapter proteins govern spread of neuropathic pain and recovery following nerve injury in male mice.

The Trif lps2 mice, which developed both ipsilateral and contralateral tactile allodynia, showed a significant increase in the ipsilateral Iba-1 activation and a numerical increase in the contralateral Iba-1 immunoreactivity that did not reach statistical significance and no difference in GFAP on either side (Figure 5 A,B,G,H).

also in 111,027 other papers