Signaling mediated by the NF-κB sub-units NF-κB1, NF-κB2 and c-Rel differentially regulate Helicobacter felis-induced gastric carcinogenesis in C57BL/6 mice.

These differences did not reach statistical significance; however, Nfkb1 −/− mice did show significantly more severe inflammatory cell infiltrates than Nfkb2 −/− mice ( Figure 3a ).

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