However, there was a clear trend towards higher levels of iNOS, more macrophages, and more iNOS producer macrophages in infected C57BL/6 mice than in infected TLR2/9 −/− mice.
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Defense against HSV-1 in a murine model is mediated by iNOS and orchestrated by the activation of TLR2 and TLR9 in trigeminal ganglia.
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