Conventional CD4+ T cells regulate IL-22-producing intestinal innate lymphoid cells.

Six weeks after depletion, both ILC numbers and ILC3 proliferation trended upwards, but differences failed to reach statistical significance ( Figure 6b-c ), while the percentage of ILC3 cells expressing MHCII fell by one third (P=0.014) ( Figure 6d ).

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Additionally, deep sequencing analysis of the16S rDNA genes of colonic commensals found that qualitative differences in bacterial repertoire between MHCII+/− and MHCII−/− genotypes did not achieve significance, as measured by principle coordinate analyses of litters ( Figure 7d ).

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