Ropivacaine attenuates endotoxin plus hyperinflation-mediated acute lung injury via inhibition of early-onset Src-dependent signaling.

Src activation, expressed as the ratio of Src, phosphorylated at tyrosine 418, over total Src (Figure 3 Aii) was on average 2-fold higher in lungs treated with LPS alone as compared to control animals, however, this effect did not reach statistical significance (n = 5, p = 1).

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