Mycobacterium leprae intracellular survival relies on cholesterol accumulation in infected macrophages: a potential target for new drugs for leprosy treatment.

Interestingly, the increased uptake of native LDL-Cho by ML-treated cells seems to be more efficiently induced by viable organisms since, although apparently higher levels were also observed with dead bacteria, the differences with the control cells did not reach statistical significance (Fig. 3 C and D).

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