In recurrent patients with high AR expression ( p = 0.023 ), there was a significant trend toward improved survival of those patients with high expression of CAMK2N1 (n = 19) compared to those with low CAMK2N1 expression (n = 20) in tumors (Figure. 1B ).
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CAMK2N1 inhibits prostate cancer progression through androgen receptor-dependent signaling.
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