Caspase-3-independent apoptotic pathways contribute to interleukin-32γ-mediated control of Mycobacterium tuberculosis infection in THP-1 cells.

Incubation of GFP- MTB -infected macrophages with the inhibitors but without addition of IL-32γ showed a trend toward increase percentage of MTB -infected cells (Figure 3 B, compare bar 1 with bars 6, 7, and 8).

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With inhibition of cathepsin L, there was also abrogation of the IL-32γ-mediated reduction in MTB that nearly reached significance (Figure 2 A).

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