Presynaptic mechanisms of lead neurotoxicity: effects on vesicular release, vesicle clustering and mitochondria number.

There were no significant differences in total mitochondria counted ( Table 1 , VC), and the mean diameter of presynaptic terminal mitochondria was similar between control and Pb 2+ -exposed groups ( Table 1 , VD), although there was a nearly significant increase in the number of mitochondria with diameter greater than 300 nm ( Table 1 , VD).

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