NKX2-5 mutations causative for congenital heart disease retain functionality and are directed to hundreds of targets.

Elife · 2015 · PMC4548209 · PMID 26146939

Europe PMC DOI

hedged sentence

0.0790

closest p · 1.6× alpha

0.0790

boldest claim

The sentences

showed a trendp = 0.079so close (0.05 < p ≤ 0.1)

NKX2-5Y191C was nuclear and, while showing no effects on myocardial progenitors or endothelial cells in three independent experiments, showed a trend towards inhibition of cardiomyocyte numbers at day 8 (∼44%; p = 0.079), suggesting that it also retained some repressive activity in this assay.

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