Cannabinoid receptor CB1 regulates STAT3 activity and its expression dictates the responsiveness to SR141716 treatment in human glioma patients' cells.

In fact, a slight difference in the degree of IFN-γ production was observed between the two different groups even though it failed to reach statistical significance (Fig. 5H ) and of note, accordingly SR14176 treatment increased susceptibility to NK cell killing of both high (Fig. 5F ) and low receptor levels cell lines (Fig. 5G ) when compared with the cytotoxicity of control untreated cells.

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