showed a trendP <0.0625
GHRHRs were more abundant in the border zones compared with their infarct zones in both GHRH‐A and placebo groups (73.5±8.1 versus 204.2±4.5 and 105.2±10.9 versus 196.3±2.3, respectively; P <0.0001 for both by Kruskal–Wallis test) ( Figure 6 B), and the GHRH‐A group showed a trend toward higher levels in the border zones ( P <0.0625 versus placebo by Wilcoxon rank test) than did the placebo group ( Figure 6 C).