Deletion of GSTA4-4 results in increased mitochondrial post-translational modification of proteins by reactive aldehydes following chronic ethanol consumption in mice.

Pathways that were nominally significant (unadjusted p -Value<0.01 in the initial comparison of 6 lists; p -Value<0.05 in the comparison of the 3 GSTA4-4 −/− exclusive lists) in at least one of the lists were retained.

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