NF-κB1, NF-κB2 and c-Rel differentially regulate susceptibility to colitis-associated adenoma development in C57BL/6 mice.

There was an increase in Tnf, Il1b , and Il6 in response to DSS, but the differences between untreated Nfkb2 −/− mice and DSS‐treated Nfkb2 −/− mice did not reach statistical significance (Figure 4 ).

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