Increased VEGF-A promotes multiple distinct aging diseases of the eye through shared pathomechanisms.

In our initial observations with a small number of mice, we had observed that VEGF‐A hyper mice lacking Il18 showed a trend toward having more CNV lesions, but the observed high variability within this group of mice and the small sample number resulted in statistically inconclusive results (Marneros, 2013 ).

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